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ChemicalBook--->CAS DataBase List--->113104-25-9

113104-25-9

113104-25-9 Structure

113104-25-9 Structure
IdentificationBack Directory
[Name]

NSC228155
[CAS]

113104-25-9
[Synonyms]

NSC228155
NSC-228155;NSC 228155
NSC228155;NSC-228155;NSC 228155
2-[(7-nitrobenzo[c][1,2,5]oxadiazol-4-yl)thio]pyridine 1-oxide
[Molecular Formula]

C11H6N4O4S
[MDL Number]

MFCD30742994
[MOL File]

113104-25-9.mol
[Molecular Weight]

290.25
Chemical PropertiesBack Directory
[Boiling point ]

627.3±65.0 °C(Predicted)
[density ]

1.72±0.1 g/cm3(Predicted)
[storage temp. ]

Keep in dark place,Sealed in dry,2-8°C
[solubility ]

≥29 mg/mL in DMSO; insoluble in H2O; insoluble in EtOH
[form ]

solid
[pka]

-1.48±0.10(Predicted)
[color ]

Light yellow to orange
Safety DataBack Directory
[Symbol(GHS) ]


GHS07
[Signal word ]

Warning
[Hazard statements ]

H302-H315-H319-H335
[Precautionary statements ]

P261-P305+P351+P338
Hazard InformationBack Directory
[Uses]

NSC 228155 is an activator of EGFR, binds to the extracellular region of EGFR and enhance tyrosine phosphorylation of EGFR[1]. NSC 228155 is also a potent inhibitor of KIX-KID interaction, inhibits kinase-inducible domain (KID) from CREB and KID-interacting domain (KIX) from CBP, with an IC50 of 0.36 μM[2].
[Biological Activity]

nsc228155 is a potent inhibitor of kix-kid interaction.cyclic-amp response-element binding protein (creb) is identified as a stimulus-activated transcription factor. its transcription activity needs its binding with creb-binding protein (cbp) after creb is phosphorylated at ser133. the domains involved for creb-cbp interaction are kinase-inducible domain (kid) from creb and kid-interacting domain (kix) from cbp.
[Synthesis]

4-Chloro-7-nitrobenzofurazan

10199-89-0

2-Pyridinethiol 1-oxide

1121-31-9

NSC228155

113104-25-9

The reaction was carried out with 4-chloro-7-nitrobenzo[c][1,2,5]oxadiazole (NBD-Cl) and 2-mercapto-pyridine-N-oxide, mixed in a 1:1 molar ratio, and stirred at room temperature for 10 min. Formation of precipitate was observed during the reaction. Subsequently, acetone (10 mL/0.1 g raw material) was added to the reaction mixture and stirring was continued. Upon completion of the reaction, the precipitate was collected by filtration through a G3 glass filter. The precipitate was further purified by preparative thin layer chromatography (TLC) using silica gel GF254 as stationary phase and a mixture of dichloromethane and methanol (10:0.2 v/v) as mobile phase, which was repeated three times. The final product was obtained as an orange-colored solid in 46% yield with a melting point of 198-199°C (literature value 201-202°C). Analysis by electrospray ionization mass spectrometry (ESI-MS) yielded the molecular ion peak [M+H]+ m/z 291, which is consistent with the calculated value of the molecular formula C11H6N4SO4 (M=290). The results of elemental analysis were as follows: calculated values C 45.52%, H 2.08%, N 19.3%, S 11.05%; measured values C 45.45%, H 2.03%, N 19.26%, S 11.01%. NMR hydrogen spectrum (1H-NMR, DMSO-d6, δ ppm, J Hz): 8.70 (d, 1H, H-6, J=7.4), 8.44 (bd, 1H, H-13, J=6.5), 8.05 (d, 1H, H-5, J=7.4), 7.39 (td, 1H, H-12, J=6.5, 3.1), 7.27- 7.22 (m, 2H, H-10, H-11). NMR carbon spectrum (13C-NMR, DMSO-d6, δ ppm): 150.73 (C-7), 146.07 (C-9), 143.47 (C-4), 136.98 (C-3), 128.52 (C-8), 138.61 (C-13), 136.63 (C-5), 131.88 (C-6), and 125.98 (C-10 or C-11), 125.84 (C-10 or C-11), 124.32 (C-12). Fourier transform infrared spectra (FT-IR, ATR, solid, ν cm-1): 3101w, 3076m, 3039w, 1518vs, 1469m, 1422s, 1363m, 1330s, 1275m, 1250s, 1227m, 1140m, 1044w, 958w, 890m, 843w, 808w. 758m, 733w, 704w.

[in vitro]

previous study found that nsc228155 could dose-dependently inhibit kix–kid interaction as measured by the split rluc assay. in living hek 293t cells, nsc228155 could inhibit creb-mediated gene transcription with an ic50 of 2.09 μm. nsc228155 also inhibited vp16-creb-mediated gene transcription with an ic50 of 6.14 μm. though this was around 3-fold higher than the ic50 of creb-mediated gene transcription, such results indicated that nsc228155 was not particularly selective in inhibiting kix–kid interaction inside these living cells. therefore, although nsc228155 was a potent inhibitor of kix-kid interaction, it was not selective against creb-mediated gene transcription, and further sar studies identified a 4-aniline substituted analog displaying a higher selectivity index [1].
[target]

EGFR

KIX-KID

0.36 μM (IC 50 )

[IC 50]

0.36 μm for kix-kid interaction
[storage]

Store at -20°C
[References]

[1] xie f, li bx, broussard c, xiao x. identification, synthesis and evaluation of substituted benzofurazans as inhibitors of creb-mediated gene transcription. bioorg med chem lett. 2013 oct 1;23(19):5371-5.
Spectrum DetailBack Directory
[Spectrum Detail]

NSC228155(113104-25-9)1HNMR
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